1. Beyond cholesterol. Modifications of low-density lipoprotein that increase
its atherogenicity. Steinberg, D.L., et al.
New England J. Med. 320:915-924 (1989).
2. Therapeutic potential of vitamin E in the pathogenesis of spontaneous
atherosclerosis. Janero, D.R., Free Radical Biol. Med. 11:129-144
(1991).
3. Oxidative injury to myocardial membrane: direct modulation by endogenous
alpha-tocopherol. Janero, D.R., et al. J. Mol. Cell. Cardiol. 21:1111-1124
(1989).
4. Hydrogen peroxide-induced oxidative stress to the mammalian heart-muscle
cell (cardiomyocyte): lethal peroxidative membrane injury. Janero, D.R.,
et al. J. Cell. Physiol. 149:347-364 (1991).
5. Therapeutic potential of vitamin E against myocardial ischemic-reperfusion
injury. Janero, D.R. Free Radical Biol. Med. 10:315-324 (1991).
6. Sequential assembly of very low density lipoprotein apolipoproteins,
triacylglycerol, and phosphoglycerides by the intact liver cell. Janero,
D.R. and Lane, M.D. J. Biol Chem. 258:14496-14504 (1983).
7. Free radical modification of low-density lipoprotein: mechanisms and
biological consequences. Heinecke, J.W. Free Radical Biol. Med. 3:65-73
(1987).
8. Malondialdehyde and thiobarbituric acid-reactivity as diagnostic indices
of lipid peroxidation and peroxidative tissue injury. Janero, D.R. Free
Radical Biol. Med. 9:515-540 (1990).
9. A receptor-mediated pathway for cholesterol homeostasis. Brown, M.S.
and Goldstein, J.L. Science232:34-47 (1986).
10. Therapeutic potential of vitamin E in the pathogenesis of spontaneous
atherosclerosis. Janero, D.R. Free Radical Biol.Med.11:129-144 (1991).
11. Protection of cardiac membrane phospholipid against oxidative injury
by calcium antagonists. Janero, D.R., et al. Biochem. Pharmacol. 21:4197-4203
(1988).
12. Therapeutic potential of vitamin E against myocardial ischemic-reperfusion
injury. Janero, D.R. Free Radical Biol. Med. 10:315-324 (1991).
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